fitting module of the origin 8.1® software Search Results


96
Vector Laboratories fitc conjugated lotus tetragonolobus lectin
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Fitc Conjugated Lotus Tetragonolobus Lectin, supplied by Vector Laboratories, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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GE Healthcare x100 software version 2 0 1
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
X100 Software Version 2 0 1, supplied by GE Healthcare, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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x100 software version 2 0 1 - by Bioz Stars, 2026-06
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Thermo Fisher fitc ebioscience 11 0441 81 hba adult hemoglobin
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
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GraphPad Software Inc non-linear fitting module in graphpad prism 6.0 software
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Non Linear Fitting Module In Graphpad Prism 6.0 Software, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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GraphPad Software Inc prism 8 enzyme kinetics-inhibition module
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Prism 8 Enzyme Kinetics Inhibition Module, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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STATA Corporation reghdfe module
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Reghdfe Module, supplied by STATA Corporation, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/reghdfe module/product/STATA Corporation
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86
Malvern Panalytical origin peak fitting module
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Origin Peak Fitting Module, supplied by Malvern Panalytical, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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GraphPad Software Inc nonlinear regression with a one site binding module
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Nonlinear Regression With A One Site Binding Module, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Sartorius AG incucytetm scratch wound cell migration software module
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Incucytetm Scratch Wound Cell Migration Software Module, supplied by Sartorius AG, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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96
Danaher Inc clampfit
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Clampfit, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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STATA Corporation seqlogit module
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Seqlogit Module, supplied by STATA Corporation, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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GraphPad Software Inc global curve fit module of prism 5.0a
Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA <t>lectin.</t> (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).
Global Curve Fit Module Of Prism 5.0a, supplied by GraphPad Software Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA lectin. (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).

Journal: iScience

Article Title: Thyroid hormone treatment counteracts cellular phenotypical remodeling in diabetic organs

doi: 10.1016/j.isci.2023.107826

Figure Lengend Snippet: Treatment with T3 counteracts pathological growth in diabetes- and glucose-injured podocytes and cardiomyocytes and maintains cardiomyocytes cytoarchitecture (A and B) Representative images of Glepp1 (A) and WT1 (B) staining in kidney serial sections of lean and diabetic animals. Quantification of individual podocyte volume and WT1-positive podocytes in lean and ZDF rats (A and B, right). (C) Quantification of Feret’s diameter in cardiomyocytes. (D and E) Evaluation of DNA content in glomerular cells (D) and cardiomyocytes (E) of control and diabetic animals. (F) Representative images of 3D reconstructed kidney (top) and heart (bottom) tissue sections. Tissues were stained with rhodamine-labeled WGA lectin. (G–I) Quantification of cell area (G, H) and axis ratio (I) in control, glucose-injured, and T3-treated human podocytes (G) and cardiomyocytes (H, I). For quantification, representative images were randomly taken from different areas of cell culture. At least 91 cells from each group were analyzed from n = 3 independent experiments (G–I). Data are expressed as mean ± SEM, one-way ANOVA corrected with Tukey’s post hoc test. ∗p < 0.05, ∗∗p < 0.01, ∗∗∗∗p < 0.0001. n = 5–6 animals per group. Scale bars, 20 μm (A, B), 40 μm (F).

Article Snippet: FITC-conjugated lotus tetragonolobus lectin (LTL, 1:80 overnight at 4°C) (Cat# FL-1321-2, Vector Laboratories) was used to stain tubular structures when needed.

Techniques: Staining, Control, Labeling, Cell Culture

Journal: iScience

Article Title: Thyroid hormone treatment counteracts cellular phenotypical remodeling in diabetic organs

doi: 10.1016/j.isci.2023.107826

Figure Lengend Snippet:

Article Snippet: FITC-conjugated lotus tetragonolobus lectin (LTL, 1:80 overnight at 4°C) (Cat# FL-1321-2, Vector Laboratories) was used to stain tubular structures when needed.

Techniques: Recombinant, Plasmid Preparation, Membrane, Enzyme-linked Immunosorbent Assay, DC Protein Assay, Software